tnrtinr
Well-Known Member
So it is just an assumption? I would rather say "I do not know" than assume.Think about it!
I do not know.
So it is just an assumption? I would rather say "I do not know" than assume.Think about it!
crack is bad. i was trying to point out that if pot was bad you'd know it. there'd be headlines "pot babies are being born everywhere. we need more incubators". i don't see that anywhere.
crack is bad. i was trying to point out that if pot was bad you'd know it. there'd be headlines "pot babies are being born everywhere. we need more incubators". i don't see that anywhere.
hey just wondering if anyone has any good links to website with info on smoking/eating marijuana while pregnant? if u have sum links that are good and from a reliable source ild love to see them!... thanks alot RIU!
i have my phd and i have multiple articles written by colleagues that you can read if you'd like to pm me with an e-mail address. they are rather technical but they are pretty solid. i'll put a few of the abstracts up now:dirtyboy... u got ur phd where?
you said smoking. what about if it is eaten?... made into cannabutter.i have my phd and i have multiple articles written by colleagues that you can read if you'd like to pm me with an e-mail address. they are rather technical but they are pretty solid. i'll put a few of the abstracts up now:
Fetal development is a period sensitive to environmental influences such as maternal drug use. The most commonly used illicit drug by pregnant women is marijuana. The present study investigated the effects of in utero marijuana exposure on expression levels of opioid-related genes in the human fetal forebrain in light of the strong interaction between the cannabinoid and opioid systems. The study group consisted of 42 midgestation fetuses from saline-induced voluntary abortions. The opioid peptide precursors (preprodynorphin and preproenkephalin (PENK)) and receptor (mu, kappa and delta) mRNA expression were assessed in distinct brain regions. The effect of prenatal cannabis exposure was analyzed by multiple regression controlling for confounding variables (maternal alcohol and cigarette use, fetal age, sex, growth measure and post-mortem interval). Prenatal cannabis exposure was significantly associated with increased mu receptor expression in the amygdala, reduced kappa receptor mRNA in mediodorsal thalamic nucleus and reduced preproenkephalin expression in the caudal putamen. Prenatal alcohol exposure primarily influenced the kappa receptor mRNA with reduced levels in the amygdala, claustrum, putamen and insula cortex. No significant effect of prenatal nicotine exposure could be discerned in the present study group. These results indicate that maternal cannabis and alcohol exposure during pregnancy differentially impair opioid-related genes in distinct brain circuits that may have long-term effects on cognitive and emotional behaviors.
here's another:
The endocannabinoid system, including its receptors (CB1 and CB2), endogenous ligands(‘endocannabinoids’, synthesising and degrading enzymes, as well as transporter molecules, has been detected from the earliest stages of embryonic development and throughout pre- and postnatal development. In addition, the endocannabinoids, notably 2-arachidonyl glycerol, are also present in maternal milk. During three distinct developmental stages (i.e. embryonic implantation, prenatal brain development and postnatal suckling), the endocannabinoid system appears to play an essential role for development and survival. Thus, during early pregnancy, successful embryonic passage through the oviduct and implantation into the uterus both require critical enzymatic control of optimal anandamide levels at the appropriate times and sites. During foetal life, the cannabinoid CB1 receptor plays a major role in brain development, regulating neural progenitor differentiation into neurones and glia and guiding axonal migration and synaptogenesis. Postnatally, CB1 receptor blockade interferes with the initiation of milk suckling in mouse pups, by inducing oral motor weakness, which exposes a critical role for CB1 receptors in the initiation of milk suckling by neonates, possibly by interfering with innervation of the tongue muscles. Manipulating the endocannabinoid system by pre- and ⁄ or postnatal administration of cannabinoids or maternal marijuana consumption, has significant, yet subtle effects on the offspring. Thus, alterations in the dopamine, GABA and endocannabioid systems have been reported while enhanced drug seeking behaviour and impaired executive (prefrontal cortical) function have also been observed. The relatively mild nature of the disruptive effects of prenatal cannabinoids may be understood in the framework of the intricate timing requirements and frequently biphasic effects of the (endo)cannabinoids. In conclusion, the endocannabinoid system plays several key roles in pre- and postnatal development. Future studies should further clarify the mechanisms involved and provide a better understanding of the adverse effects of prenatal exposure, in order to design strategies for the treatment of conditions such as infertility, mental retardation and failure-to-thrive.
you can also just use common sense. during pregnancy the fetus is 100% linked to the mother. anything that goes into the mother is filtered through the ambiotic fluid and into the child. smoking marijuana puts carcinogens into the body... and thus into the baby. hope that helps.
I just don't like reading a bunch of medical terminology without references to back it up....It is just a bunch of words to me until I know who wrote it and what their credentials are....
I'm sure after you misscarry it's real easy!! loli hear smoking meth while pregnant helps you stay thin.
I just don't like reading a bunch of medical terminology without references to back it up....It is just a bunch of words to me until I know who wrote it and what their credentials are....
i respect anyone who wants to take a closer look at anyone of these articles. that's great. read the articles and make your own judgements.And it’s still a bunch of words even with sources. The subjects are regulated to the control subject on any research project. I don’t see anything about that. Sounds like a bunch of bias (resrched by the tobacco industry) bull shiz.
I am on both of the pages now, thanks.i respect anyone who wants to take a closer look at anyone of these articles. that's great. read the articles and make your own judgements.
1st abstract: Journal of Neuroendocrinology; May 2008 Supplement, Vol. 20, p75-81, 7p, 1 chart, 2 graphs
2nd abstract: Pharmacogenomics Journal; 2006, Vol. 6 Issue 4, p255-264, 10p, 3 charts, 3 graphs
those are both cutting edge medical journals that are not "biases researched by the tobacco industry". they are peer reviewed by the top medical professionals in the world. check out the journal and read up on the subject. its fascinating.